Cardiovascular effects of methamphetamine in Parkinson's disease patients
Identifieur interne : 003D87 ( Main/Exploration ); précédent : 003D86; suivant : 003D88Cardiovascular effects of methamphetamine in Parkinson's disease patients
Auteurs : Nicola Pavese [Royaume-Uni] ; Ornella Rimoldi [Royaume-Uni] ; Alexander Gerhard [Royaume-Uni] ; David J. Brooks [Royaume-Uni] ; Paola Piccini [Royaume-Uni]Source :
- Movement Disorders [ 0885-3185 ] ; 2004-03.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Homme.
English descriptors
- KwdEn :
- Adrenergic Fibers (drug effects), Analysis of Variance, Blood Pressure (drug effects), Brain (drug effects), Catecholamines (antagonists & inhibitors), Dopamine Agents (administration & dosage), Dopamine Agents (adverse effects), Dopamine Agents (therapeutic use), Drug Therapy, Combination, Electrocardiography (drug effects), Female, Heart Rate (drug effects), Human, Humans, Injections, Intravenous, Levodopa (therapeutic use), Male, Methamphetamine (administration & dosage), Methamphetamine (adverse effects), Methamphetamine (therapeutic use), Middle Aged, Myocardium (metabolism), Nervous system diseases, Parkinson Disease (drug therapy), Parkinson disease, Parkinson's disease, Presynaptic Terminals (drug effects), Psychomotor Agitation (etiology), Tomography, Emission-Computed, blood pressure, methamphetamine, postganglionic sympathetic neurons.
- MESH :
- chemical , administration & dosage : Dopamine Agents, Methamphetamine.
- chemical , adverse effects : Dopamine Agents, Methamphetamine.
- chemical , antagonists & inhibitors : Catecholamines.
- drug effects : Adrenergic Fibers, Blood Pressure, Brain, Electrocardiography, Heart Rate, Presynaptic Terminals.
- drug therapy : Parkinson Disease.
- etiology : Psychomotor Agitation.
- metabolism : Myocardium.
- chemical , therapeutic use : Dopamine Agents, Levodopa, Methamphetamine.
- Analysis of Variance, Drug Therapy, Combination, Female, Humans, Injections, Intravenous, Male, Middle Aged, Tomography, Emission-Computed.
Abstract
Cardiovascular responses after intravenous methamphetamine were assessed in 11 Parkinson's disease (PD) patients. Systolic blood pressure (SBP), diastolic blood pressure (DBP), heart rate (HR), and electrocardiogram (ECG) were monitored for 103 minutes. After methamphetamine administration, SBP and DBP increased significantly in both PD and normal controls whereas placebo had no effect. In PD patients, however, the duration of SBP and DBP responses to methamphetamine and the maximum increase from baseline was attenuated compared with the controls. A significant correlation was found between individual BP responsiveness and the Unified Parkinson's Disease Rating Scale (UPDRS) motor score. These findings suggest that in PD there is impairment of catecholamine release from peripheral sympathetic presynaptic terminals, which correlates with motor impairment. © 2003 Movement Disorder Society
Url:
DOI: 10.1002/mds.10651
Affiliations:
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Le document en format XML
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<series><title level="j">Movement Disorders</title>
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<term>Brain (drug effects)</term>
<term>Catecholamines (antagonists & inhibitors)</term>
<term>Dopamine Agents (administration & dosage)</term>
<term>Dopamine Agents (adverse effects)</term>
<term>Dopamine Agents (therapeutic use)</term>
<term>Drug Therapy, Combination</term>
<term>Electrocardiography (drug effects)</term>
<term>Female</term>
<term>Heart Rate (drug effects)</term>
<term>Human</term>
<term>Humans</term>
<term>Injections, Intravenous</term>
<term>Levodopa (therapeutic use)</term>
<term>Male</term>
<term>Methamphetamine (administration & dosage)</term>
<term>Methamphetamine (adverse effects)</term>
<term>Methamphetamine (therapeutic use)</term>
<term>Middle Aged</term>
<term>Myocardium (metabolism)</term>
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<term>Psychomotor Agitation (etiology)</term>
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<term>postganglionic sympathetic neurons</term>
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<front><div type="abstract" xml:lang="en">Cardiovascular responses after intravenous methamphetamine were assessed in 11 Parkinson's disease (PD) patients. Systolic blood pressure (SBP), diastolic blood pressure (DBP), heart rate (HR), and electrocardiogram (ECG) were monitored for 103 minutes. After methamphetamine administration, SBP and DBP increased significantly in both PD and normal controls whereas placebo had no effect. In PD patients, however, the duration of SBP and DBP responses to methamphetamine and the maximum increase from baseline was attenuated compared with the controls. A significant correlation was found between individual BP responsiveness and the Unified Parkinson's Disease Rating Scale (UPDRS) motor score. These findings suggest that in PD there is impairment of catecholamine release from peripheral sympathetic presynaptic terminals, which correlates with motor impairment. © 2003 Movement Disorder Society</div>
</front>
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<name sortKey="Piccini, Paola" sort="Piccini, Paola" uniqKey="Piccini P" first="Paola" last="Piccini">Paola Piccini</name>
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